48 research outputs found
Exploring the X-ray and γ-ray properties of the redback millisecond pulsar PSR J1723-2837
published_or_final_versio
Orbital-phase-dependent γ-ray Emissions from the Black Widow Pulsar
published_or_final_versio
Search for pulsed γ-ray emission from globular cluster M28
published_or_final_versio
Discovery of X-ray Pulsation from the Geminga-like Pulsar PSR J 2021+4026
published_or_final_versio
A Detailed X-Ray Investigation of PSR J2021+4026 and the γ-Cygni Supernova Remnant
published_or_final_versio
Radio emission from Supernova Remnants
The explosion of a supernova releases almost instantaneously about 10^51 ergs
of mechanic energy, changing irreversibly the physical and chemical properties
of large regions in the galaxies. The stellar ejecta, the nebula resulting from
the powerful shock waves, and sometimes a compact stellar remnant, constitute a
supernova remnant (SNR). They can radiate their energy across the whole
electromagnetic spectrum, but the great majority are radio sources. Almost 70
years after the first detection of radio emission coming from a SNR, great
progress has been achieved in the comprehension of their physical
characteristics and evolution. We review the present knowledge of different
aspects of radio remnants, focusing on sources of the Milky Way and the
Magellanic Clouds, where the SNRs can be spatially resolved. We present a brief
overview of theoretical background, analyze morphology and polarization
properties, and review and critical discuss different methods applied to
determine the radio spectrum and distances. The consequences of the interaction
between the SNR shocks and the surrounding medium are examined, including the
question of whether SNRs can trigger the formation of new stars. Cases of
multispectral comparison are presented. A section is devoted to reviewing
recent results of radio SNRs in the Magellanic Clouds, with particular emphasis
on the radio properties of SN 1987A, an ideal laboratory to investigate
dynamical evolution of an SNR in near real time. The review concludes with a
summary of issues on radio SNRs that deserve further study, and analyzing the
prospects for future research with the latest generation radio telescopes.Comment: Revised version. 48 pages, 15 figure
Acute hunger does not always undermine prosociality
This is the final version. Available on open access from Nature Research via the DOI in this recordData Availability: The data that support the findings of this paper are available on the OSF website
(https://osf.io/zexd7/?view_only=480593713c904397a033e751a6da7a69).It has been argued that, when they are acutely hungry, people act in self-protective ways
by keeping resources to themselves rather than sharing them. In four studies, using experimental,
quasi-experimental, and correlational designs (total N = 795), we examine the effects of acute
hunger on prosociality in a wide variety of non-interdependent tasks (e.g. dictator game) and
interdependent tasks (e.g. public goods games). While our procedures successfully increase
subjective hunger and decrease blood glucose, we do not find significant effects of hunger on
prosociality. This is true for both decisions incentivized with money and with food. Metaanalysis across all tasks reveals a very small effect of hunger on prosociality in noninterdependent tasks (d = .108), and a non-significant effect in interdependent tasks (d = -0.076).
In study five (N = 197), we show that, in stark contrast to our empirical findings, people hold
strong lay theories that hunger undermines prosociality.Volkswagen Foundatio
A genetic cause of Alzheimer disease: mechanistic insights from Down syndrome
Down syndrome, caused by an extra copy of chromosome 21, is associated with a greatly increased risk of early onset Alzheimer disease. It is thought that this risk is conferred by the presence of three copies of the gene encoding amyloid precursor protein (APP), an Alzheimer risk factor, although the possession of extra copies of other chromosome 21 genes may also play a role. Further study of the mechanisms underlying the development of Alzheimer disease in Down syndrome could provide insights into the mechanisms that cause dementia in the general population